Petre BM, Attar, S. Osteology (Bone Anatomy). . eMedicine from WebMD. Updated June 23, 2011. Available at:

Petre BM, Grutter PW, Rose DM, Belkoff SM, McFarland EG, Petersen SA. TricepTriceps tendons: a biomechanical comparison of intact and repaired strength.J Shoulder Elbow Surg. 2011 Mar;20(2):213-8. Epub 2010 Dec 10.

Petre BM, Deune E. Metacarpophalangeal and Interphalangeal Ligaments. eMedicine from WebMD. Updated November 24, 2010. Available at:

Lashuel HA. Hartley DM. Petre BM. Wall JS. Simon MN. Walz T. Lansbury PT Jr. Mixtures of wild-type and a pathogenic (E22G) form of Abeta40 in vitro accumulate protofibrils, including amyloid pores. Journal of Molecular Biology. 332(4):795-808.

Lashuel HA. Petre BM. Wall J. Simon M. Nowak RJ. Walz T. Lansbury PT Jr. Alpha-synuclein, especially the Parkinson’s disease-associated mutants, forms pore-like annular and tubular protofibrils. Journal of Molecular Biology. 322(5):1089-102.

Takagi J. Petre BM. Walz T. Springer TA. Global conformational rearrangements in integrin extracellular domains in outside-in and inside-out signaling. Cell. 110(5):599-11.

Muller M. Yong M. Peng XH. Petre B. Arora S. Ambudkar SV. Evidence for the role of glycosylation in accessibility of the extracellular domains of human MRP1 (ABCC1). Biochemistry. 41(31):10123-32.

Lashuel HA. Hartley D. Petre BM. Walz T. Lansbury PT Jr. Neurodegenerative disease: amyloid pores from pathogenic mutations. Nature. 418(6895):291.

Cascio P. Call M. Petre BM. Walz T. Goldberg AL. Properties of the hybrid form of the 26S proteasome containing both 19S and PA28 complexes. EMBO Journal. 21(11):2636-45.

Lederkremer GZ. Cheng Y. Petre BM. Vogan E. Springer S. Schekman R. Walz T. Kirchhausen T. Structure of the Sec23p/24p and Sec13p/31p complexes of COPII. Proceedings of the National Academy of Sciences of the United States of America. 98(19):10704-9.